Dihydroartemisinin and gefitinib synergistically inhibit NSCLC cell growth and promote apoptosis via the Akt/mTOR/STAT3 pathway-Reference-Cited by-同舟云学术

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Dihydroartemisinin and gefitinib synergistically inhibit NSCLC cell growth and promote apoptosis via the Akt/mTOR/STAT3 pathway

Published:2017-03 Issue:3 Volume:16 Page:3475-3481
ISSN:1791-2997
Container-title:Molecular Medicine Reports
language:en
Short-container-title:

Author:

Jin Hong,Jiang Ai-Ying,Wang Han,Cao Yong,Wu Yan,Jiang Xiao-Feng

Publisher

Spandidos Publications

Subject

Cancer Research,Oncology,Genetics,Molecular Biology,Molecular Medicine,Biochemistry

Cited by 37 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Dihydroartemisinin induces ferroptosis in T cell acute lymphoblastic leukemia cells by downregulating SLC7A11 and activating the ATF4‑CHOP signaling pathway;Oncology Letters;2024-05-24

2. YES1 as a potential target to overcome drug resistance in EGFR-deregulated non-small cell lung cancer;Archives of Toxicology;2024-03-05

3. Dihydroartemisinin Enhances the Effects of Oxaliplatin by Activating PDCD5/ARAF mediated Signal Transduction in Colon Cancer;2023-09-20

4. Chemoresistance Mechanisms in Non-Small Cell Lung Cancer—Opportunities for Drug Repurposing;Applied Biochemistry and Biotechnology;2023-09-18

5. Dihydroartemisinin enhances gefitinib cytotoxicity against lung adenocarcinoma cells by inducing ROS‐dependent apoptosis and ferroptosis;The Kaohsiung Journal of Medical Sciences;2023-04-14

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