Inhibition of cytoskeletal protein carbonylation may protect against oxidative damage in traumatic brain injury
Author:
Publisher
Spandidos Publications
Subject
Cancer Research,Immunology and Microbiology (miscellaneous),General Medicine
Cited by 8 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Ceramide‐mediated orchestration of oxidative stress response through filopodia‐derived small extracellular vesicles;Journal of Extracellular Vesicles;2024-07
2. Neuroanatomical zones of human traumatic brain injury reveal significant differences in protein profile and protein oxidation: Implications for secondary injury events;Journal of Neurochemistry;2023-09-11
3. Differentiated Neurons Are More Vulnerable to Organophosphate and Carbamate Neurotoxicity than Undifferentiated Neurons Due to the Induction of Redox Stress and Accumulate Oxidatively-Damaged Proteins;Brain Sciences;2023-04-26
4. Cadmium-induced toxicity in Meretrix meretrix ovary is characterized by oxidative damage with changes in cell morphology and apoptosis-related factors;Frontiers in Marine Science;2022-12-01
5. β-Actin: An Emerging Biomarker in Ischemic Stroke;Cellular and Molecular Neurobiology;2022-05-13
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