Cholesterol biosynthesis inhibitor RO 48‑8071 inhibits pancreatic ductal adenocarcinoma cell viability by deactivating the JNK and ERK/MAPK signaling pathway-Reference-Cited by-同舟云学术

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Cholesterol biosynthesis inhibitor RO 48‑8071 inhibits pancreatic ductal adenocarcinoma cell viability by deactivating the JNK and ERK/MAPK signaling pathway

Published:2021-09-28 Issue:6 Volume:24 Page:
ISSN:1791-2997
Container-title:Molecular Medicine Reports
language:
Short-container-title:Mol Med Rep

Author:

Ding Zhen¹,Gu Yanan²,Huang Dake³,Zhou Hong²,Zhu Tingting²,Luo Xin²,Zhang Sumei²,Zhang Shengquan²,Qian Yeben¹

Affiliation:

1. Department of General Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230022, P.R. China

2. Department of Biochemistry and Molecular Biology, Anhui Medical University, Hefei, Anhui 230032, P.R. China

3. Comprehensive Laboratory, School of Basic Medical Sciences, Anhui Medical University, Hefei, Anhui 230032, P.R. China

Publisher

Spandidos Publications

Subject

Cancer Research,Oncology,Genetics,Molecular Biology,Molecular Medicine,Biochemistry

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Lipid metabolism reprogramming in renal cell carcinomas;Medical Oncology;2024-09-06

2. Lanosterol synthase loss of function decreases the malignant phenotypes of HepG2 cells by deactivating the Src/MAPK signaling pathway;Oncology Letters;2023-05-23

3. Should oxidosqualene cyclase in the cholesterol biosynthetic pathway be considered an anti-cancer target?;Frontiers in Cell and Developmental Biology;2022-12-13

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