Ferroptosis contributes to hemolytic hyperbilirubinemia‑induced brain damage in vivo and in vitro

Author:

Zhou Jinfu1,Lin Xinpei1,Liao Sining1,Li Guilin1,Tang Jianping1,Luo Jinying2,Zhang Chenran1,Wu Siying3,Xu Liangpu4,Li Huangyuan1

Affiliation:

1. Department of Preventive Medicine, School of Public Health, Fujian Medical University, Fuzhou, Fujian 350122, P.R. China

2. Department of Obstetrics and Gynecology, Fujian Maternity and Child Health Hospital, College of Clinical Medicine for Obstetrics & Gynecology and Pediatrics, Fujian Medical University, Fuzhou, Fujian 350001, P.R. China

3. Fujian Provincial Key Laboratory of Environmental Factors and Cancer, School of Public Health, Fujian Medical University, Fuzhou, Fujian 350122, P.R. China

4. Department of Medical Genetic Diagnosis and Therapy Center, Fujian Maternity and Child Health Hospital, College of Clinical Medicine for Obstetrics & Gynecology and Pediatrics, Fujian Medical University, Fuzhou, Fujian 350001, P.R. China

Publisher

Spandidos Publications

Subject

Cancer Research,Oncology,Genetics,Molecular Biology,Molecular Medicine,Biochemistry

Reference76 articles.

1. Hyperbilirubinemia in the Newborn;Lauer;Pediatr Rev,2011

2. Bilirubin: The toxic mechanisms of an antioxidant molecule;Soto Conti;Arch Argent Pediatr,2021

3. Acute neonatal bilirubin encephalopathy in the State of Utah 2009–2018;Christensen;Blood Cells Mol Dis,2018

4. Neonatal hemolysis and risk of bilirubin-induced neurologic dysfunction;Wong;Semin Fetal Neonatal Med,2015

5. Childhood neurodevelopmental outcomes of survivors of acute bilirubin encephalopathy: A retrospective cohort study;Kumar;Early Hum Dev,2021

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