Interleukin 32 participates in cardiomyocyte‑induced oxidative stress, inflammation and apoptosis during hypoxia/reoxygenation via the NOD2/NOX2/MAPK signaling pathway-Reference-Cited by-同舟云学术

Interleukin 32 participates in cardiomyocyte‑induced oxidative stress, inflammation and apoptosis during hypoxia/reoxygenation via the NOD2/NOX2/MAPK signaling pathway

Published:2022-07-12 Issue:3 Volume:24 Page:
ISSN:1792-0981
Container-title:Experimental and Therapeutic Medicine
language:
Short-container-title:Exp Ther Med

Author:

Li Yuanyuan¹,Wang Zhongyan²

Affiliation:

1. Department of Cardiovascular Surgery, The Second Hospital of Tianjin Medical University, Tianjin 300211, P.R. China

2. Department of Geriatrics, The Second Hospital of Tianjin Medical University, Tianjin 300211, P.R. China

Publisher

Spandidos Publications

Subject

Cancer Research,Immunology and Microbiology (miscellaneous),General Medicine

Reference36 articles.

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1. Hypoxia-Induced Myocardial Hypertrophy Companies with Apoptosis Enhancement and p38-MAPK Pathway Activation;High Altitude Medicine & Biology;2024-09-01

2. Induction of IL‐32 in the immune response of keratinocytes to Mycobacterium marinum infection;Experimental Dermatology;2023-06-13