β-Amyrin Ameliorates Alzheimer's Disease-Like Aberrant Synaptic Plasticity in the Mouse Hippocampus-Reference-Cited by-同舟云学术

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β-Amyrin Ameliorates Alzheimer's Disease-Like Aberrant Synaptic Plasticity in the Mouse Hippocampus

Published:2020-01-01 Issue:1 Volume:28 Page:74-82
ISSN:1976-9148
Container-title:Biomolecules & Therapeutics
language:en
Short-container-title:Biomolecules & Therapeutics

Author:

Park Hye Jin¹,Kwon Huiyoung¹,Lee Ji Hye²,Cho Eunbi¹,Lee Young Choon¹³,Moon Minho⁴,Jun Mira³⁵,Kim Dong Hyun¹³,Jung Ji Wook⁶

Affiliation:

1. Department of Medicinal Biotechnology, College of Health Sciences, Dong-A University, Busan 49315, Republic of Korea

2. Division of Endocrinology, School of Medicine, Kyungpook National University, Daegu 41944, Republic of Korea

3. Institute of Convergence Bio-Health, Dong-A University, Busan 49315, Republic of Korea

4. Department of Biochemistry, College of Medicine, Konyang University, Daejeon 35365, Republic of Korea

5. Department of Food Science and Nutrition, College of Health Sciences, Dong-A University, Busan 49315, Republic of Korea

6. Department of Herbal Medicinal Pharmacology, College of Herbal Bio-industry, Daegu Haany University, Kyungsan 38610, Republic of Korea

Funder

Korea Small and Medium Business Administration

Publisher

The Korean Society of Applied Pharmacology

Subject

Drug Discovery,Pharmacology,Molecular Medicine,Biochemistry

Link

http://pdf.medrang.co.kr/BT2/2020/028/BT028-01-07.pdf

Reference49 articles.

1. Minocycline prevents the development of depression-like behavior and hippocampal inflammation in a rat model of Alzheimer’s disease

2. Soluble oligomeric amyloid-β induces calcium dyshomeostasis that precedes synapse loss in the living mouse brain

3. Glycogen synthase kinase-3 (GSK3): Regulation, actions, and diseases

4. Calcium flux-independent NMDA receptor activity is required for Aβ oligomer-induced synaptic loss

5. Amyloid-β interrupts the PI3K-Akt-mTOR signaling pathway that could be involved in brain-derived neurotrophic factor-induced Arc expression in rat cortical neurons

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