Decreased Interaction of Raf-1 with Its Negative Regulator Spry2 as a Mechanism for Acquired Drug Resistance
Author:
Publisher
The Korean Society of Applied Pharmacology
Subject
Drug Discovery,Pharmacology,Molecular Medicine,Biochemistry
Link
http://ocean.kisti.re.kr/downfile/crosscheck/ksp/JAKO201115952329169.pdf
Reference17 articles.
1. The Raf/MEK/ERK pathway can govern drug resistance, apoptosis and sensitivity to targeted therapy
2. The enhancement of Raf-1 kinase activity by knockdown of Spry2 is associated with high sensitivity to paclitaxel in v-Ha-ras-transformed NIH 3T3 fibroblasts
3. Interaction of tamoxifen with the multidrug resistance P-glycoprotein
4. RKIP Sensitizes Prostate and Breast Cancer Cells to Drug-induced Apoptosis
5. Reversal effect of Raf-1/Mdr-1 siRNAs co-transfection on multidrug resistance in KBv200 cell line
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1. ATG5 knockout promotes paclitaxel sensitivity in drug-resistant cells via induction of necrotic cell death;The Korean Journal of Physiology & Pharmacology;2020-05-01
2. Induction of Resistance to BRAF Inhibitor Is Associated with the Inability of Spry2 to Inhibit BRAF-V600E Activity in BRAF Mutant Cells;Biomolecules & Therapeutics;2015-07-31
3. Defective autophagy in multidrug resistant cells may lead to growth inhibition by BH3-mimetic gossypol;Journal of Cellular Physiology;2013-03-21
4. Differential inhibitory effects of two Raf-targeting drugs, sorafenib and PLX4720, on the growth of multidrug-resistant cells;Molecular and Cellular Biochemistry;2012-09-02
5. Src Family Kinase Inhibitor PP2 Induces LC3 Conversion in a Manner That is Uncoupled from Autophagy and Increases Apoptosis in Multidrug-Resistant Cells;Biomolecules and Therapeutics;2012-07-31
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