Insulin as a Potent Stimulator of Akt, ERK and Inhibin-βE Signaling in Osteoblast-Like UMR-106 Cells-Reference-Cited by-同舟云学术

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Insulin as a Potent Stimulator of Akt, ERK and Inhibin-βE Signaling in Osteoblast-Like UMR-106 Cells

Published:2016-11-01 Issue:6 Volume:24 Page:589-594
ISSN:2005-4483
Container-title:Biomolecules & Therapeutics
language:en
Short-container-title:Biomolecules & Therapeutics

Author:

Ramalingam Mahesh¹,Kwon Yong-Dae²,Kim Sung-Jin¹

Affiliation:

1. Department of Pharmacology and Toxicology, Metabolic Diseases Research Laboratory, Kyung Hee University, Seoul 02447, Republic of Korea

2. Department of Oral and Maxillofacial Surgery, School of Dentistry, Kyung Hee University, Seoul 02447, Republic of Korea

Funder

Kyung Hee University(Kyunghee University)

National Research Foundation of Korea(NRF)

Ministry of Education, Science and Technology(Korean Ministry of Education, Science and Technology)

Publisher

The Korean Society of Applied Pharmacology

Subject

Drug Discovery,Pharmacology,Molecular Medicine,Biochemistry

Link

http://pdf.medrang.co.kr/BT2/2016/024/BT024-06-04.pdf

Reference26 articles.

1. Cross-talk between mitogenic Ras/MAPK and survival PI3K/Akt pathways: a fine balance

2. Inhibin/activin-betaE subunit in normal and malignant human cervical tissue and cervical cancer cell lines

3. Hydrogen peroxide-mediated phosphorylation of ERK1/2, Akt/PKB and JNK in cortical neurones: dependence on Ca2+ and PI3-kinase

4. Phosphoinositide-3-OH kinase-dependent regulation of glycogen synthase kinase 3 and protein kinase B/AKT by the integrin-linked kinase

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