Characteristics of gastric cancer detected within 1 year after successful eradication of Helicobacter pylori-Reference-Cited by-同舟云学术

Characteristics of gastric cancer detected within 1 year after successful eradication of Helicobacter pylori

Published:2016 Issue:3 Volume:59 Page:226-230
ISSN:0912-0009
Container-title:Journal of Clinical Biochemistry and Nutrition
language:en
Short-container-title:J. Clin. Biochem. Nutr.

Author:

Nishizawa Toshihiro¹²³,Suzuki Hidekazu⁴,Arano Toru¹⁵,Yoshida Shuntaro¹⁶,Yamashita Hiroharu¹⁷,Hata Keisuke¹⁸,Kanai Takanori³,Yahagi Naohisa²,Toyoshima Osamu¹

Affiliation:

1. Gastroenterology, Toyoshima Endoscopy Clinic

2. Division of Research and Development for Minimally Invasive Treatment, Cancer Center, Keio University School of Medicine

3. Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine

4. Medical Education Center, Keio University School of Medicine

5. Department of Gastroenterology, Toshiba General Hospital

6. Department of Gastroenterology, Graduate School of Medicine, The University of Tokyo

7. Department of Gastrointestinal Surgery, Graduate School of Medicine, The University of Tokyo

8. Department of Surgical Oncology, Graduate School of Medicine, The University of Tokyo

Publisher

The Society for Free Radical Research Japan

Subject

Clinical Biochemistry,Nutrition and Dietetics,Medicine (miscellaneous)

Link

https://www.jstage.jst.go.jp/article/jcbn/59/3/59_16-43/_pdf

Reference30 articles.

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2. 2 Nishizawa T, Suzuki H. Gastric carcinogenesis and underlying molecular mechanisms: Helicobacter pylori and novel targeted therapy. Biomed Res Int 2015; 2015: 794378.

3. 3 Suzuki H, Mori H. Helicobacter pylori: Helicobacter pylori gastritis--a novel distinct disease entity. Nat Rev Gastroenterol Hepatol 2015; 12: 556–557.

4. 4 Handa O, Naito Y, Yoshikawa T. Helicobacter pylori: a ROS-inducing bacterial species in the stomach. Inflamm Res 2010; 59: 997–1003.

5. 5 Nguyen TT, Kim SJ, Park JM, Hahm KB, Lee HJ. Repressed TGF-β signaling through CagA-Smad3 interaction as pathogenic mechanisms of Helicobacter pylori-associated gastritis. J Clin Biochem Nutr 2015; 57: 113–120.

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