Dietary zinc deficiency induces oxidative stress and promotes tumor necrosis factor-α- and interleukin-1β-induced RANKL expression in rat bone
Author:
Affiliation:
1. Department of Nutritional Science, Faculty of Applied Bioscience, Tokyo University of Agriculture
Publisher
The Society for Free Radical Research Japan
Subject
Clinical Biochemistry,Nutrition and Dietetics,Medicine (miscellaneous)
Link
https://www.jstage.jst.go.jp/article/jcbn/58/2/58_15-87/_pdf
Reference47 articles.
1. 1 Prasad AS. Discovery of human zinc deficiency: its impact on human health and disease. Adv Nutr 2013; 4: 176–190.
2. 2 Iwata K, Enomoto H, Nishiguchi S, et al. Serum zinc value in patients with hepatitis virus-related chronic liver disease: association with the histological degree of liver fibrosis and with the severity of varices in compensated cirrhosis. J Clin Biochem Nutr 2014; 55: 147–152.
3. 3 Vallee BL, Falchuk KH. The biochemical basis of zinc physiology. Physiol Rev 1993; 73: 79–118.
4. 4 Holloway WR, Collier FM, Herbst RE, Hodge JM, Nicholson GC. Osteoblast-mediated effects of zinc on isolated rat osteoclast: Inhibition of bone resorption and enhancement of osteoclast number. Bone 1996; 19: 137–142.
5. 5 Togari A, Arakawa S, Arai M, Matsumoto S. Alteration of in vitro bone metabolism and tooth formation by zinc. Gen Pharmacol 1993; 24: 1133–1140.
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