Mitophagy-mediated inflammation and oxidative stress contribute to muscle wasting in cancer cachexia
Author:
Affiliation:
1. Department of General Surgery/Shanghai Clinical Nutrition Research Center, Zhongshan Hospital, Fudan University
Publisher
The Society for Free Radical Research Japan
Subject
Clinical Biochemistry,Nutrition and Dietetics,Medicine (miscellaneous)
Link
https://www.jstage.jst.go.jp/article/jcbn/73/1/73_23-1/_pdf
Reference30 articles.
1. 1 Fearon K, Strasser F, Anker SD, et al. Definition and classification of cancer cachexia: an international consensus. Lancet Oncol 2011; 12: 489–495.
2. 2 Schmidt SF, Rohm M, Herzig S, Berriel Diaz M. Cancer cachexia: more than skeletal muscle wasting. Trends Cancer 2018; 4: 849–860.
3. 3 Anandavadivelan P, Lagergren P. Cachexia in patients with oesophageal cancer. Nat Rev Clin Oncol 2016; 13: 185–198.
4. 4 Zhang Y, Wang J, Wang X, et al. The autophagic-lysosomal and ubiquitin proteasome systems are simultaneously activated in the skeletal muscle of gastric cancer patients with cachexia. Am J Clin Nutr 2020; 111: 570–579.
5. 5 Tardif N, Klaude M, Lundell L, Thorell A, Rooyackers O. Autophagic-lysosomal pathway is the main proteolytic system modified in the skeletal muscle of esophageal cancer patients. Am J Clin Nutr 2013; 98: 1485–1492.
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1. Exploring heterogeneity: a dive into preclinical models of cancer cachexia;American Journal of Physiology-Cell Physiology;2024-08-01
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