Maternal high-fructose consumption provokes placental oxidative stress resulting in asymmetrical fetal growth restriction in rats
Author:
Affiliation:
1. Department of Nutrition and Food Hygiene, School of Public Health, Qingdao University
Publisher
The Society for Free Radical Research Japan
Subject
Clinical Biochemistry,Nutrition and Dietetics,Medicine (miscellaneous)
Link
https://www.jstage.jst.go.jp/article/jcbn/69/1/69_21-19/_pdf
Reference36 articles.
1. 1 Regnault TR, Gentili S, Sarr O, Toop CR, Sloboda DM. Fructose, pregnancy and later life impacts. Clin Exp Pharmacol Physiol 2013; 40: 824–837.
2. 2 Barker DJ. The origins of the developmental origins theory. J Intern Med 2007; 261: 412–417.
3. 3 Yang Y, Kan H, Yu X, Yang Y, Li L, Zhao M. Relationship between dietary inflammatory index, hs-CRP level in the second trimester and neonatal birth weight: a cohort study. J Clin Biochem Nutr 2020; 66: 163–167.
4. 4 Wong AC, Ko CW. Carbohydrate intake as a risk factor for biliary sludge and stones during pregnancy. J Clin Gastroenterol 2013; 47: 700–705.
5. 5 Alzamendi A, Del Zotto H, Castrogiovanni D, Romero J, Giovambattista A, Spinedi E. Oral metformin treatment prevents enhanced insulin demand and placental dysfunction in the pregnant rat fed a fructose-rich diet. ISRN Endocrinol 2012; 2012: 757913.
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