Activation of Anoctamin-1 Limits Pulmonary Endothelial Cell Proliferation via p38–Mitogen-activated Protein Kinase–Dependent Apoptosis-Reference-Cited by-同舟云学术

Activation of Anoctamin-1 Limits Pulmonary Endothelial Cell Proliferation via p38–Mitogen-activated Protein Kinase–Dependent Apoptosis

Published:2018-05 Issue:5 Volume:58 Page:658-667
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Allawzi Ayed M.¹²,Vang Alexander¹,Clements Richard T.¹³,Jhun Bong Sook⁴,Kue Nouaying R.¹,Mancini Thomas J.¹,Landi Amy K.⁴,Terentyev Dmitry⁴,O-Uchi Jin⁴,Comhair Suzy A.⁵,Erzurum Serpil C.⁵,Choudhary Gaurav¹⁶

Affiliation:

1. Vascular Research Laboratory, Providence Veterans Affairs Medical Center, Providence, Rhode Island

2. Department of Molecular Pharmacology, Physiology, and Biotechnology, Brown University, Providence, Rhode Island

3. Department of Surgery and

4. Department of Medicine, Cardiovascular Research Center, Rhode Island Hospital and Alpert Medical School of Brown University, Providence, Rhode Island

5. Lerner Research Institute, Cleveland Clinic, Cleveland Ohio; and

6. Department of Medicine, Warren Alpert Medical School of Brown University, Providence, Rhode Island

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Link

http://www.atsjournals.org/doi/pdf/10.1165/rcmb.2016-0344OC

Reference27 articles.

1. New Molecular Targets of Pulmonary Vascular Remodeling in Pulmonary Arterial Hypertension

2. Expression of angiogenesis-related molecules in plexiform lesions in severe pulmonary hypertension: evidence for a process of disordered angiogenesis

3. Endothelial Dysfunction in Pulmonary Hypertension

4. Alterations of cellular bioenergetics in pulmonary artery endothelial cells

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