Identification of Drug Candidates to Suppress Cigarette Smoke–induced Inflammation via Connectivity Map Analyses-Reference-Cited by-同舟云学术

Identification of Drug Candidates to Suppress Cigarette Smoke–induced Inflammation via Connectivity Map Analyses

Published:2018-06 Issue:6 Volume:58 Page:727-735
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Vanderstocken Gilles¹,Dvorkin-Gheva Anna¹²,Shen Pamela¹,Brandsma Corry-Anke³,Obeidat Ma’en⁴,Bossé Yohan⁵⁶^ORCID,Hassell John A.²,Stampfli Martin R.¹⁷^ORCID

Affiliation:

1. Department of Pathology and Molecular Medicine, McMaster Immunology Research Centre

2. Department of Pathology and Molecular Medicine, Centre for Functional Genomics, and

3. Research Institute for Asthma and COPD, University Medical Center Groningen, University of Groningen, Groningen, the Netherlands

4. The University of British Columbia Centre for Heart Lung Innovation, St. Paul’s Hospital, Vancouver, British Columbia, Canada

5. Centre de Recherche de l’Institut Universitaire de Cardiologie et de Pneumologie de Québec, Québec City, Québec, Canada; and

6. Department of Molecular Medicine, Laval University, Québec City, Québec, Canada

7. Department of Medicine, Firestone Institute for Respiratory Health at St. Joseph’s Healthcare, McMaster University, Hamilton, Ontario, Canada

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Link

https://www.atsjournals.org/doi/pdf/10.1165/rcmb.2017-0202OC

Reference49 articles.

1. The Neutrophil in Chronic Obstructive Pulmonary Disease. Too Little, Too Late or Too Much, Too Soon?

2. The Connectivity Map: Using Gene-Expression Signatures to Connect Small Molecules, Genes, and Disease

3. Gene expression signature-based chemical genomic prediction identifies a novel class of HSP90 pathway modulators

4. A Drug Repositioning Approach Identifies Tricyclic Antidepressants as Inhibitors of Small Cell Lung Cancer and Other Neuroendocrine Tumors

5. Molecular mechanisms underlying variations in lung function: a systems genetics analysis

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