Interplay between EZH2 and G9a Regulates CXCL10 Gene Repression in Idiopathic Pulmonary Fibrosis-Reference-Cited by-同舟云学术

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Interplay between EZH2 and G9a Regulates CXCL10 Gene Repression in Idiopathic Pulmonary Fibrosis

Published:2018-04 Issue:4 Volume:58 Page:449-460
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Coward William R.¹²,Brand Oliver J.¹²,Pasini Alice¹²³,Jenkins Gisli¹²,Knox Alan J.¹²,Pang Linhua¹²

Affiliation:

1. Division of Respiratory Medicine and

2. Nottingham Respiratory Research Unit, University of Nottingham, City Hospital, Nottingham, United Kingdom; and

3. Department of Electrical, Electronic and Information Engineering “Guglielmo Marconi” (DEI), University of Bologna, Cesena, Italy

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Link

http://www.atsjournals.org/doi/pdf/10.1165/rcmb.2017-0286OC

Reference41 articles.

1. Landmark approvals in idiopathic pulmonary fibrosis

2. The Myofibroblast

3. Increased constitutive αSMA and Smad2/3 expression in idiopathic pulmonary fibrosis myofibroblasts is KCa3.1-dependent

4. Defective Histone Acetylation Is Responsible for the Diminished Expression of Cyclooxygenase 2 in Idiopathic Pulmonary Fibrosis

5. A central role for G9a and EZH2 in the epigenetic silencing of cyclooxygenase‐2 in idiopathic pulmonary fibrosis

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