Lack of IL-1 Receptor Signaling Reduces Spontaneous Airway Eosinophilia in Juvenile Mice with Muco-Obstructive Lung Disease-Reference-Cited by-同舟云学术

Lack of IL-1 Receptor Signaling Reduces Spontaneous Airway Eosinophilia in Juvenile Mice with Muco-Obstructive Lung Disease

Published:2020-03 Issue:3 Volume:62 Page:300-309
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Brown Ryan¹,Paulsen Michelle¹,Schmidt Simone¹,Schatterny Jolanthe¹,Frank Angela¹,Hirtz Stephanie¹,Delaney Rebecca²,Doherty Declan²,Hagner Matthias¹,Taggart Cliff²,Weldon Sinéad²,Mall Marcus A.¹³⁴^ORCID

Affiliation:

1. Department of Translational Pulmonology, Translational Lung Research Centre Heidelberg, German Center for Lung Research, University of Heidelberg, Heidelberg, Germany

2. Airway Innate Immunity Research Group, Wellcome-Wolfson Institute for Experimental Medicine, School of Medicine, Dentistry and Biomedical Sciences, Queen’s University Belfast, Belfast, Northern Ireland

3. Department of Pediatric Pulmonology, Immunology and Intensive Care Medicine, Charité–Universitätsmedizin Berlin, Berlin, Germany; and

4. Berlin Institute of Health, Berlin, Germany

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Link

https://www.atsjournals.org/doi/pdf/10.1165/rcmb.2018-0359OC

Reference40 articles.

1. Eosinophilic airway inflammation: role in asthma and chronic obstructive pulmonary disease

2. Eosinophils as diagnostic tools in chronic lung disease

3. Eosinophilic Inflammation in Allergic Asthma

4. Subepithelial immunopathology of the large airways in smokers with and without chronic obstructive pulmonary disease

5. Eosinophilic Inflammation in Stable Chronic Obstructive Pulmonary Disease

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