Pulmonary Innate Immune Dysfunction in Human Immunodeficiency Virus
Author:
Affiliation:
1. Division of Pulmonary, Allergy, Critical Care, and Sleep Medicine, Emory University School of Medicine, Atlanta, Georgia; and
2. the Atlanta Veterans Administration Medical Center, Decatur, Georgia
Publisher
American Thoracic Society
Subject
Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology
Link
http://www.atsjournals.org/doi/pdf/10.1165/rcmb.2016-0213TR
Reference54 articles.
1. Immune Reconstitution duringPneumocystisLung Infection: Disruption of Surfactant Component Expression and Function byS-Nitrosylation
2. Pulmonary surfactant in patients with Pneumocystis pneumonia and acquired immunodeficiency syndrome
3. Surfactant Protein A Modulates the Inflammatory Response in Macrophages during Tuberculosis
4. Surfactant Protein A Binds to HIV and Inhibits Direct Infection of CD4+Cells, but Enhances Dendritic Cell-Mediated Viral Transfer
5. Surfactant protein D binds to human immunodeficiency virus (HIV) envelope protein gp120 and inhibits HIV replication
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