The mTORC2/SGK1/NDRG1 Signaling Axis Is Critical for the Mesomesenchymal Transition of Pleural Mesothelial Cells and the Progression of Pleural Fibrosis-Reference-Cited by-同舟云学术

The mTORC2/SGK1/NDRG1 Signaling Axis Is Critical for the Mesomesenchymal Transition of Pleural Mesothelial Cells and the Progression of Pleural Fibrosis

Published:2024-01 Issue:1 Volume:70 Page:50-62
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Keshava Shiva¹,Owens Shuzi¹,Qin Wenyi¹,Jeffers Ann²,Kyei Perpetual³,Komatsu Satoshi¹,Kleam Joshua¹,Ikebe Mitsuo¹,Idell Steven²¹,Tucker Torry A.²¹

Affiliation:

1. Department of Cellular and Molecular Biology, and

2. Texas Lung Injury Institute,

3. Biotechnology Graduate Program, The University of Texas Health Science Center at Tyler, Tyler, Texas

Funder

National Heart, Lung, and Blood Institute

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Link

https://www.atsjournals.org/doi/pdf/10.1165/rcmb.2023-0131OC

Reference55 articles.

1. Mesomesenchymal transition of pleural mesothelial cells is PI3K and NF-κB dependent

2. Pathogenesis of pleural fibrosis

3. Biomechanical Force and Cellular Stiffness in Lung Fibrosis

4. The Role of the Mammalian Target of Rapamycin (mTOR) in Pulmonary Fibrosis

5. Plasminogen-Plasmin System in the Pathogenesis and Treatment of Lung and Pleural Injury

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2. Inhibition of the rapamycin-insensitive mTORC1 /4E-BP1 axis attenuates TGF-β1-induced fibrotic response in human Tenon's fibroblasts;Experimental Eye Research;2024-07

3. Pleural Fibrosis: Now That’s What mTORC(ing) About;American Journal of Respiratory Cell and Molecular Biology;2024-01

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