TBK1 Is Required for Host Defense Functions Distinct from Type I IFN Expression and Myeloid Cell Recruitment in Murine<i>Streptococcus pneumoniae</i>Pneumonia-Reference-Cited by-同舟云学术

TBK1 Is Required for Host Defense Functions Distinct from Type I IFN Expression and Myeloid Cell Recruitment in MurineStreptococcus pneumoniaePneumonia

Published:2022-06 Issue:6 Volume:66 Page:671-681
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Hagan Robert S.¹²^ORCID,Gomez John C.²³,Torres-Castillo Jose¹²,Martin Jessica R.²³,Doerschuk Claire M.¹²³

Affiliation:

1. Division of Pulmonary Diseases and Critical Care Medicine, Department of Medicine,

2. Marsico Lung Institute, and

3. Center for Airways Disease, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina

Funder

National Heart, Lung, and Blood Institute

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Link

https://www.atsjournals.org/doi/pdf/10.1165/rcmb.2020-0311OC

Reference26 articles.

1. Neutrophils in Homeostasis, Immunity, and Cancer

2. Type I interferons in infectious disease

3. Recent insights into the complexity of Tank-binding kinase 1 signaling networks: The emerging role of cellular localization in the activation and substrate specificity of TBK1

4. Loss of Tbk1 kinase activity protects mice from diet-induced metabolic dysfunction

5. The kinase TBK1 controls IgA class switching by negatively regulating noncanonical NF-κB signaling

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