Indole-3-Acetic Acid Protects Against Lipopolysaccharide-induced Endothelial Cell Dysfunction and Lung Injury through the Activation of USP40
Author:
Affiliation:
1. Department of Physiology and Cell Biology, Dorothy M. Davis Heart and Lung Research Institute, and
2. Department of Internal Medicine, The Ohio State University, Columbus, Ohio
Funder
Foundation for the National Institutes of Health
Publisher
American Thoracic Society
Link
https://www.atsjournals.org/doi/pdf/10.1165/rcmb.2024-0159OC
Reference54 articles.
1. Endothelial-Dependent Mechanisms of Leukocyte Recruitment to the Vascular Wall
2. Pulmonary Microvascular Albumin Leak Is Associated with Endothelial Cell Death in Murine Sepsis-Induced Lung Injury In Vivo
3. The effects of sepsis on endothelium and clinical implications
4. Broken Barriers: A New Take on Sepsis Pathogenesis
5. Endothelial activation and dysfunction in COVID-19: from basic mechanisms to potential therapeutic approaches
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. The Plant Hormone Indole-3-Acetic Acid Helps the Endothelial Barrier Seal after Lung Injury;American Journal of Respiratory Cell and Molecular Biology;2024-09
2. Escherichia coli Nissle 1917 Protects against Sepsis-Induced Intestinal Damage by Regulating the SCFA/GPRs Signaling Pathway;Microorganisms;2024-08-08
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