Inhibition of ABCC1 Decreases cAMP Egress and Promotes Human Airway Smooth Muscle Cell Relaxation-Reference-Cited by-同舟云学术

Inhibition of ABCC1 Decreases cAMP Egress and Promotes Human Airway Smooth Muscle Cell Relaxation

Published:2022-01 Issue:1 Volume:66 Page:96-106
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Cao Gaoyuan¹,Lam Hong¹,Jude Joseph A.¹,Karmacharya Nikhil¹,Kan Mengyuan²,Jester William¹,Koziol-White Cynthia¹³,Himes Blanca E.²,Chupp Geoffrey L.⁴,An Steven S.¹³^ORCID,Panettieri Reynold A.¹^ORCID

Affiliation:

1. Rutgers Institute for Translational Medicine and Science, and

2. Department of Biostatistics, Epidemiology and Informatics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania; and

3. Department of Pharmacology, Rutgers Robert Wood Johnson Medical School, Rutgers, The State University of New Jersey, New Brunswick, New Jersey;

4. Division of Pulmonary, Critical Care and Sleep Medicine, School of Medicine, Yale University, New Haven, Connecticut

Funder

New Jersey Alliance for Clinical and Translational Science–National Center for Advancing Translational Sciences

National Heart, Lung, and Blood Institute

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Reference50 articles.

1. New drugs for asthma

2. Do Biophysical Properties of the Airway Smooth Muscle in Culture Predict Airway Hyperresponsiveness?

3. An inflammation-independent contraction mechanophenotype of airway smooth muscle in asthma

4. The small heat shock-related protein, HSP20, is a cAMP-dependent protein kinase substrate that is involved in airway smooth muscle relaxation

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