Multiplatform Single-Cell Analysis Identifies Immune Cell Types Enhanced in Pulmonary Fibrosis-Reference-Cited by-同舟云学术

Multiplatform Single-Cell Analysis Identifies Immune Cell Types Enhanced in Pulmonary Fibrosis

Published:2022-07 Issue:1 Volume:67 Page:50-60
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Serezani Ana P. M.¹^ORCID,Pascoalino Bruno D.¹,Bazzano Julia M. R.¹,Vowell Katherine N.¹,Tanjore Harikrishna¹,Taylor Chase J.¹,Calvi Carla L.¹,McCall A. Scott¹,Bacchetta Matthew D.²,Shaver Ciara M.¹,Ware Lorraine B.¹³^ORCID,Salisbury Margaret L.¹^ORCID,Banovich Nicholas E.⁴,Kendall Peggy L.⁵,Kropski Jonathan A.¹⁶⁷^ORCID,Blackwell Timothy S.¹⁶⁷

Affiliation:

1. Division of Allergy, Pulmonary, and Critical Care Medicine, Department of Medicine,

2. Department of Thoracic Surgery, and

3. Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee;

4. Translational Genomics Research Institute, Phoenix, Arizona;

5. Division of Allergy and Immunology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri;

6. Department of Cell and Developmental Biology, Vanderbilt University, Nashville, Tennessee; and

7. Department of Veterans Affairs Medical Center, Nashville, Tennessee

Funder

Boehringer Ingelheim

Doris Duke Charitable Foundation

National Heart, Lung, and Blood Institute

U.S. Department of Defense

Department of Veterans Affairs

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Link

https://www.atsjournals.org/doi/pdf/10.1165/rcmb.2021-0418OC

Reference80 articles.

1. Inflammation and immunity in IPF pathogenesis and treatment

2. GRP78 and CHOP modulate macrophage apoptosis and the development of bleomycin-induced pulmonary fibrosis

3. Macrophage Akt1 Kinase-Mediated Mitophagy Modulates Apoptosis Resistance and Pulmonary Fibrosis

4. Blockade of IL-6 Trans Signaling Attenuates Pulmonary Fibrosis

5. Identification of an atypical monocyte and committed progenitor involved in fibrosis

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