AKT Phosphorylates FAM13A and Promotes Its Degradation via CUL4A/DDB1/DCAF1 E3 Complex
Author:
Affiliation:
1. Channing Division of Network Medicine, Brigham and Women’s Hospital, Boston, Massachusetts;
2. Harvard Medical School, Boston, Massachusetts; and
3. Department of Pathology, Beth Israel Deaconess Medical Center, Boston, Massachusetts
Funder
National Institutes of Health
Publisher
American Thoracic Society
Subject
Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology
Link
https://www.atsjournals.org/doi/pdf/10.1165/rcmb.2022-0362OC
Reference47 articles.
1. Chronic obstructive pulmonary disease
2. Global, regional, and national deaths, prevalence, disability-adjusted life years, and years lived with disability for chronic obstructive pulmonary disease and asthma, 1990–2015: a systematic analysis for the Global Burden of Disease Study 2015
3. Potential etiologic and functional implications of genome-wide association loci for human diseases and traits
4. Variants in FAM13A are associated with chronic obstructive pulmonary disease
5. Genetic loci associated with chronic obstructive pulmonary disease overlap with loci for lung function and pulmonary fibrosis
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