Surfactant-Associated Protein B Is Critical to Survival in Nickel-Induced Injury in Mice-Reference-Cited by-同舟云学术

Surfactant-Associated Protein B Is Critical to Survival in Nickel-Induced Injury in Mice

Published:2009-08 Issue:2 Volume:41 Page:226-236
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Bein Kiflai,Wesselkamper Scott C.,Liu Xiangdong,Dietsch Maggie,Majumder Nilanjana,Concel Vincent J.,Medvedovic Mario,Sartor Maureen A.,Henning Lisa N.,Venditto Carmen,Borchers Michael T.,Barchowsky Aaron,Weaver Timothy E.,Tichelaar Jay W.,Prows Daniel R.,Korfhagen Thomas R.,Hardie William D.,Bachurski Cindy J.,Leikauf George D.

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Reference52 articles.

1. Acute Lung Injury and the Acute Respiratory Distress Syndrome

2. Epidemiology and Outcomes of Acute Lung Injury

3. Lippmann M, Ito K, Hwang JS, Maciejczyk P, Chen LC. Cardiovascular effects of nickel in ambient air.Environ Health Perspect2006;114:1662–1669.

4. Inhalational Nickel Carbonyl Poisoning in Waste Processing Workers

5. Quantitative Trait Analysis of Nickel-Induced Acute Lung Injury in Mice

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