Expression of JP-8–Induced Inflammatory Genes in AEII Cells Is Mediated by NF-κB and PARP-1-Reference-Cited by-同舟云学术

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Expression of JP-8–Induced Inflammatory Genes in AEII Cells Is Mediated by NF-κB and PARP-1

Published:2006-10 Issue:4 Volume:35 Page:479-487
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Espinoza Luis A.,Tenzin Fnu,Cecchi Andrea O.,Chen Zun,Witten Mark L.,Smulson Mark E.

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Reference44 articles.

1. Proliferation during early phases of bronchiolar repair in neonatal rabbits following lung injury by 4-ipomeanol

2. Hays AM, Lantz RC, Witten ML. Correlation between in vivo and in vitro pulmonary responses to jet propulsion fuel-8 using precision-cut lung slices and a dynamic organ culture system.Toxicol Pathol2003;31:200–207.

3. Altered Expression of γ-Synuclein and Detoxification-Related Genes in Lungs of Rats Exposed to JP-8

4. Roles of Oxidative Stress and Glutathione Depletion in JP-8 Jet Fuel-Induced Apoptosis in Rat Lung Epithelial Cells

5. Mechanisms of JP-8 Jet Fuel Toxicity. I. Induction of Apoptosis in Rat Lung Epithelial Cells

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3. Cytochrome P450 2A13 is an efficient enzyme in metabolic activation of aflatoxin G1 in human bronchial epithelial cells;Archives of Toxicology;2013-08-02

4. Impairment of alveolar type-II cells involved in the toxicity of Aflatoxin G1 in rat lung;Food and Chemical Toxicology;2012-09

5. Benzo[a]pyrene and tumor necrosis factor-α coordinately increase genotoxic damage and the production of proinflammatory mediators in alveolar epithelial type II cells;Toxicology Letters;2011-10

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