Glucagon release dependent on and independent of changes in cytosolic Ca2+: Studies using Ca2+-clamped rat pancreatic islets
Author:
Affiliation:
1. Third Department of Internal Medicine, University of Nagoya, School of Medicine
2. Department of Internal Medicine, Aichigakuin University, School of Dentistry
Publisher
Biomedical Research Press
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
https://www.jstage.jst.go.jp/article/biomedres/7/4/7_291/_pdf
Cited by 8 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Inhibition by nitric oxide of Ca2+ responses in rat pancreatic α-cells;Life Sciences;2002-05
2. Adrenaline Stimulates Glucagon Secretion in Pancreatic A-Cells by Increasing the Ca2+ Current and the Number of Granules Close to the L-Type Ca2+ Channels;Journal of General Physiology;1997-09-01
3. Oscillatory Ca2+ signaling in somatostatin-producing cells from the human pancreas;Metabolism;1997-04
4. Suppression of Ca2+ oscillations in glucagon-producing α2-cells by insulin/glucose and amino acids;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;1996-02
5. Regulation of glucagon release from pancreatic A-cells;Biochemical Pharmacology;1991-06
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