Exposure to valproic acid during middle to late-stage corticogenesis induces learning and social behavioral abnormalities with attention deficit/hyperactivity in adult mice
Author:
Affiliation:
1. Laboratory of Molecular Biology, Department of Biofunctional Analysis, Gifu Pharmaceutical University
Publisher
Biomedical Research Press
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
https://www.jstage.jst.go.jp/article/biomedres/40/5/40_179/_pdf
Reference44 articles.
1. 1 American Psychiatric Association (2013) Diagnostic and Statistical Manual of Mental Disorders (DSM-5). 5th edn., VA: American Psychiatric Association, Arlington.
2. 2 Auger ML and Floresco SB (2014) Prefrontal cortical GABA modulation of spatial reference and working memory. Int J Neuropsychopharmacol 18, pyu013.
3. 3 Beck SL (2001) Does genomic imprinting contribute to valproic acid teratogenicity? Reprod Toxicol 15, 43–48.
4. 4 Bell SB and DeWall N (2018) Does transcranial direct current stimulation to the prefrontal cortex affect social behavior? A meta-analysis. Soc Cogn Affect Neurosci 13, 899–906.
5. 5 Betancur C (2011) Etiological heterogeneity in autism spectrum disorders: more than 100 genetic and genomic disorders and still counting. Brain Res 1380, 42–77.
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