Characteristics of podocyte injury in malignant hypertensive nephropathy of rats (MSHRSP/Kpo strain)
Author:
Affiliation:
1. Department of Pharmacology, Faculty of Medicine, Kinki University
2. Department of Pathology, Faculty of Medicine, Kinki University
3. Life Science Research Institute, Kinki University
Publisher
Biomedical Research Press
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
https://www.jstage.jst.go.jp/article/biomedres/36/5/36_313/_pdf
Reference32 articles.
1. 1 Barisoni L and Mundel P (2003) Podocyte biology and the emerging understanding of podocyte diseases. Am J Nephrol 23, 353–360.
2. 2 D’Agati VD (2008) The spectrum of focal segmental glomerulosclerosis: new insights. Curr Opin Nephrol Hypertens 17, 271–281.
3. 3 Dasgupta I, Porter C, Innes A and Burden R (2007) “Benign”hypertensive nephrosclerosis. QJM 100, 113–119.
4. 4 Durvasula RV, Petermann AT, Hiromura K. Blonski M, Pippin J, Mundel P, Pichler R, Griffin S, Couser WG and Shankland SJ (2004) Activation of a local tissue angiotensin system in podocytes by mechanical strain. Kidney Int 65, 30–39.
5. 5 Finne K, Vethe H, Skogstrand T, Leh S, Dahl TD, Tenstad O, Berven FS, Reed RK and Vikse BE (2014) Proteomic analysis of formalin-fixed paraffin-embedded glomeruli suggests depletion of glomerular filtration barrier proteins in two-kidney, one-clip hypertensive rats. Nephrol Dial Transplant 29, 2217–2227.
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