COPPER METABOLISM IN THE MACULAR MOUSE, AN ANIMAL MODEL FOR MENKES’KINKY HAIR DISEASE: CHANGES IN THE CYTOCHROME C OXIDASE ACTIVITY AND COPPER CONTENT IN VARIOUS ORGANS AFTER COPPER ADMINISTRATION -Reference-Cited by-同舟云学术

COPPER METABOLISM IN THE MACULAR MOUSE, AN ANIMAL MODEL FOR MENKES’KINKY HAIR DISEASE: CHANGES IN THE CYTOCHROME C OXIDASE ACTIVITY AND COPPER CONTENT IN VARIOUS ORGANS AFTER COPPER ADMINISTRATION

Published:1993 Issue:1 Volume:14 Page:57-64
ISSN:0388-6107
Container-title:Biomedical Research
language:en
Short-container-title:Biomed. Res.

Author:

KOYAMA MASAHIKO¹,HORIIKE KIHACHIRO²,NOZAKI MITSUHIRO²,SHIMADA MORIMI¹

Affiliation:

1. Departments of Pediatrics, Shiga University of Medical Science

2. Departments of Biochemistry, Shiga University of Medical Science

Publisher

Biomedical Research Press

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

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1. Decreased Expression of the Slc31a1 Gene and Cytoplasmic Relocalization of Membrane CTR1 Protein in Renal Epithelial Cells: A Potent Protective Mechanism against Copper Nephrotoxicity in a Mouse Model of Menkes Disease;International Journal of Molecular Sciences;2022-09-28

2. Physarum polycephalum—a new take on a classic model system;Journal of Physics D: Applied Physics;2017-09-15

3. Mutation in the CPC motif-containing 6th transmembrane domain affects intracellular localization, trafficking and coppertransport efficiency of ATP7Aprotein in mosaic mutant mice––an animal model of Menkes disease;Metallomics;2012

4. Alternative splicing in the Atp7a gene in the Cu deficient mosaic mutation in mice;Folia Biologica;2004-12-01

5. Pathological structure of the kidney from adult mice with mosaic mutation;Journal of Inherited Metabolic Disease;2003-02