Calcineurin Aβ gene knockdown inhibits transient outward potassium current ion channel remodeling in hypertrophic ventricular myocyte
Author:
Affiliation:
1. Department of Cardiology, Guizhou Provincial People’s Hospital , No. 83 Zhongshandong Road , Guiyang 550002 , China
2. Department of Cardiology, The Affiliated Hospital of Guizhou Medical University , Guiyang 550025 , China
Abstract
Publisher
Walter de Gruyter GmbH
Subject
General Agricultural and Biological Sciences,General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,General Neuroscience
Link
https://www.degruyter.com/document/doi/10.1515/biol-2021-0107/pdf
Reference32 articles.
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2. Shimizu I, Minamino T. Physiological and pathological cardiac hypertrophy. J Mol Cell Cardiol. 2016;97:245–62.
3. Huang D, Hua W, Fang Q, Yan J, Su Y, Liu B, et al. POSCD-China Biventricular pacemaker and defibrillator implantation in patients with chronic heart failure in China. ESC Heart Fail. 2021;8(1):546–54.
4. Wang Y, Hill JA. Electrophysiological remodeling in heart failure. J Mol Cell Cardiol. 2010;48(4):619–32.
5. Kepenek ES, Ozcinar E, Tuncay E, Akcali KC, Akar AR, Turan B. Differential expression of genes participating in cardiomyocyte electrophysiological remodeling via membrane ionic mechanisms and Ca2+-handling in human heart failure. Mol Cell Biochem. 2020;463(1–2):33–44.
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