GABAergic Deafferentation Hypothesis of Brain Aging and Alzheimer's Disease; Pharmacologic Profile of the Benzodiazepine Antagonist, Flumazenil-Reference-Cited by-同舟云学术

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GABAergic Deafferentation Hypothesis of Brain Aging and Alzheimer's Disease; Pharmacologic Profile of the Benzodiazepine Antagonist, Flumazenil

Published:1995-01 Issue:3 Volume:6 Page:
ISSN:2191-0200
Container-title:Reviews in the Neurosciences
language:
Short-container-title:

Author:

Marczynski Thaddens J.

Publisher

Walter de Gruyter GmbH

Subject

General Neuroscience

Link

https://www.degruyter.com/document/doi/10.1515/REVNEURO.1995.6.3.221/pdf

Reference138 articles.

1. Specific changes in local cerebral glucose utilization in the rat brain induced by acute and chronic diazepam

2. Increased .GAMMA.-aminobutyrate aminotransferase activity in brain of patients with Alzheimer's disease.

3. Dopamine uptake sites in Parkinson's disease and in dementia of the Alzheimer type

4. Basic fibroblast growth factor prevents death of lesioned cholinergic neurons in vivo

5. Interchangeability of Stress and Amphetamine in Sensitization

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1. The GABAergic system in Alzheimer’s disease: a systematic review with meta-analysis;Molecular Psychiatry;2023-07-07

2. Benzodiazepines and Related Drugs as a Risk Factor in Alzheimer's Disease Dementia;Frontiers in Aging Neuroscience;2020-01-08

3. Dissecting Complex and Multifactorial Nature of Alzheimer’s Disease Pathogenesis: a Clinical, Genomic, and Systems Biology Perspective;Molecular Neurobiology;2015-09-09

4. GABAergic neurotransmission and new strategies of neuromodulation to compensate synaptic dysfunction in early stages of Alzheimerâ€™s disease;Frontiers in Cellular Neuroscience;2014-06-25

5. In vivo molecular imaging of the GABA/benzodiazepine receptor complex in the aged rat brain;Neurobiology of Aging;2012-07

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