Ascorbate-Dependent Capacity of Dialysed Rat Liver Cytosol to Prevent Nonenzymatic Lipid Peroxidation
Author:
Publisher
Walter de Gruyter GmbH
Subject
Biochemistry
Link
https://www.degruyter.com/document/doi/10.1515/bchm3.1992.373.2.1111/pdf
Reference20 articles.
1. Thiyl free radicals: direct observations of electron transfer reactions with phenothiazines and ascorbate
2. Autoxidation of micelles and model membranes. Quantitative kinetic measurements can be made by using either water-soluble or lipid-soluble initiators with water-soluble or lipid-soluble chain-breaking antioxidants
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2. Cytoprotection of vascular endotheliocytes by phosphorylated ascorbate through suppression of oxidative stress that is generated immediately after post-anoxic reoxygenation or with alkylhydroperoxides;Journal of Cellular Biochemistry;2004-10-18
3. Anti-apoptotic defense ofbcl-2 gene against hydroperoxide-induced cytotoxicity together with suppressed lipid peroxidation, enhanced ascorbate uptake, and upregulated Bcl-2 protein;Journal of Cellular Biochemistry;2003-04-15
4. Dual Role of Plasma Membrane Electron Transport Systems in Defense;Critical Reviews in Biochemistry and Molecular Biology;2000-01
5. Ascorbate-Fe2+ lipid-peroxidation of rat liver microsomes: Effect of vitamin E and cytosolic proteins;Molecular and Cellular Biochemistry;1998
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