Application of Hypoxia-Induced Shut Down of Replicon Initiation to the Analysis of Replication Intermediates in Ehrlich Ascites Cells
Author:
Publisher
Walter de Gruyter GmbH
Subject
Biochemistry
Link
https://www.degruyter.com/document/doi/10.1515/bchm3.1986.367.2.1209/pdf
Reference10 articles.
1. Replication of Eukaryotic Chromosomes: A Close-up of the Replication Fork
2. Reversible inhibition of replicon initiation in Ehrlich ascites cells by anaerobiosis
3. Oxygen dependence of nuclear DNA replication in Ehrlich ascites cells
4. Use of zonal centrifugation for preparing synchronous cultures from Ehrlich ascites cells grown in vivo
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1. PD184352 Releases the Regular Hypoxic Reversible DNA Replication Arrest in T24 Cells;BMB Reports;2007-11-30
2. Cdk2 activity is dispensable for triggering replicon initiation after transient hypoxia in T24 cells;FEBS Journal;2005-11
3. Analyzing changes of chromatin-bound replication proteins occurring in response to and after release from a hypoxic block of replicon initiation in T24 cells;European Journal of Biochemistry;2003-09-09
4. Detection of a sub-set of polysomal mRNAs associated with modulation of hypusine formation at the G1-S boundary Proposal of a role for eIF-5A in onset of DNA replication;FEBS Letters;1995-06-12
5. Reversible shutdown of replicon initiation by transient hypoxia in Ehrlich ascites cells. Dependence of initiation on short-lived protein;European Journal of Biochemistry;1992-12
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