Ephedrine Alkaloid-Independent High-Affinity Immunoglobulin-E Receptor (FcεRI) Internalization Results in CCL2 Production without Inducing Mast Cell Degranulation-Reference-Cited by-同舟云学术

Ephedrine Alkaloid-Independent High-Affinity Immunoglobulin-E Receptor (FcεRI) Internalization Results in CCL2 Production without Inducing Mast Cell Degranulation

Published:2023-06-01 Issue:6 Volume:46 Page:811-816
ISSN:0918-6158
Container-title:Biological and Pharmaceutical Bulletin
language:en
Short-container-title:Biological & Pharmaceutical Bulletin

Author:

Nagata Yuka¹,Sasaki Yohei²,Suzuki Ryo¹

Affiliation:

1. Laboratory of Hygienic Chemistry, Faculty of Pharmaceutical Sciences, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University

2. Laboratory of Pharmacognosy, Faculty of Pharmaceutical Sciences, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University

Publisher

Pharmaceutical Society of Japan

Subject

Pharmaceutical Science,Pharmacology,General Medicine

Link

https://www.jstage.jst.go.jp/article/bpb/46/6/46_b23-00006/_pdf

Reference28 articles.

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2. 2) Burton OT, Oettgen HC. Beyond immediate hypersensitivity: evolving roles for IgE antibodies in immune homeostasis and allergic diseases. Immunol. Rev., 242, 128–143 (2011).

3. 3) Gomez G. Current strategies to inhibit high affinity FcεRI-mediated signaling for the treatment of allergic disease. Front. Immunol., 10, 175 (2019).

4. 4) Kalesnikoff J, Galli SJ. New developments in mast cell biology. Nat. Immunol., 9, 1215–1223 (2008).

5. 5) Yin L, Li X, Hou J. Macrophages in periodontitis: a dynamic shift between tissue destruction and repair. Jpn. Dent. Sci. Rev., 58, 336–347 (2022).

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