Involvement of Cannabinoid Receptors and Adenosine A2B Receptor in Enhanced Migration of Lung Cancer A549 Cells Induced by γ-Ray Irradiation
Author:
Affiliation:
1. Department of Radiation Biosciences, Faculty of Pharmaceutical Sciences, Tokyo University of Science
2. Department of Medicinal and Life Sciences, Faculty of Pharmaceutical Sciences, Tokyo University of Science
Publisher
Pharmaceutical Society of Japan
Subject
Pharmaceutical Science,Pharmacology,General Medicine
Link
https://www.jstage.jst.go.jp/article/bpb/47/1/47_b23-00631/_pdf
Reference43 articles.
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2. 2) Nishimaki N, Tsukimoto M, Kitami A, Kojima S. Autocrine regulation of γ-irradiation-induced DNA damage response via extracellular nucleotides-mediated activation of P2Y6 and P2Y12 receptors. DNA Repair (Amst.), 11, 657–665 (2012).
3. 3) Di Virgilio F, Adinolfi E. Extracellular purines, purinergic receptors and tumor growth. Oncogene, 36, 293–303 (2017).
4. 4) Alvarez CL, Troncoso MF, Espelt MV. Extracellular ATP and adenosine in tumor microenvironment: roles in epithelial-mesenchymal transition, cell migration, and invasion. J. Cell. Physiol., 237, 389–400 (2022).
5. 5) Kitabatake K, Yoshida E, Kaji T, Tsukimoto M. Involvement of adenosine A2B receptor in radiation-induced translocation of epidermal growth factor receptor and DNA damage response leading to radioresistance in human lung cancer cells. Biochim. Biophys. Acta, Gen. Subj., 1864, 129457 (2020).
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