The Inhibitory G Protein Gi Identified as Pertussis Toxin-Catalyzed ADP-Ribosylation
Author:
Affiliation:
1. Department of Physiological Chemistry, Graduate School of Pharmaceutical Sciences, The University of Tokyo
Publisher
Pharmaceutical Society of Japan
Subject
Pharmaceutical Science,Pharmacology,General Medicine
Link
https://www.jstage.jst.go.jp/article/bpb/35/12/35_b212024/_pdf
Reference53 articles.
1. 1) Katada T, Ui M. Accelerated turnover of blood glucose in pertussis-sensitized rats due to combined actions of endogenous insulin and adrenergic β-stimulation. Biochim. Biophys. Acta, 421, 57–69 (1976).
2. 2) Yajima M, Hosoda K, Kanbayashi Y, Nakamura T, Nogimori K, Mizushima Y, Nakase Y, Ui M. Islets-activating protein (IAP) in Bordetella pertussis that potentiates insulin secretory responses of rats. Purification and characterization. J. Biochem., 83, 295–303 (1978).
3. 3) Yajima M, Hosoda K, Kanbayashi Y, Nakamura T, Takahashi I, Ui M. Biological properties of islets-activating protein (IAP) purified from the culture medium of Bordetella pertussis. J. Biochem., 83, 305–312 (1978).
4. 4) Katada T, Ui M. Effect of in vivo pretreatment of rats with a new protein purified from Bordetella pertussis on in vitro secretion of insulin: role of calcium. Endocrinology, 104, 1822–1827 (1979).
5. 5) Katada T, Ui M. Islet-activating protein. Enhanced insulin secretion and cyclic AMP accumulation in pancreatic islets due to activation of native calcium ionophores. J. Biol. Chem., 254, 469–479 (1979).
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