eIF2α-Independent Inhibition of TNF-α-Triggered NF-κB Activation by Salubrinal
Author:
Affiliation:
1. Department of Molecular Signaling, Interdisciplinary Graduate School of Medicine and Engineering, University of Yamanashi
2. Department of Organ Regulatory Surgery and Department of Advanced Cancer Immunotherapy, Fukushima Medical University
Publisher
Pharmaceutical Society of Japan
Subject
Pharmaceutical Science,Pharmacology,General Medicine
Link
https://www.jstage.jst.go.jp/article/bpb/38/9/38_b15-00312/_pdf
Reference31 articles.
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2. 2) Lee SK, Kim YS. Phosphorylation of eIF2α attenuates statin-induced apoptosis by inhibiting the stabilization and translocation of p53 to the mitochondria. Int. J. Oncol., 42, 810–816 (2013).
3. 3) Kessel D. Protection of Bcl-2 by salubrinal. Biochem. Biophys. Res. Commun., 346, 1320–1323 (2006).
4. 4) Yokota H, Hamamura K, Chen A, Dodge TR, Tanjung N, Abedinpoor A, Zhang P. Effects of salubrinal on development of osteoclasts and osteoblasts from bone marrow-derived cells. BMC Musculoskelet. Disord., 14, 197 (2013).
5. 5) Huang X, Chen Y, Zhang H, Ma Q, Zhang YW, Xu H. Salubrinal attenuates β-amyloid-induced neuronal death and microglial activation by inhibition of the NF-κB pathway. Neurobiol. Aging, 33, 1007.e9–1007.e17 (2012).
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