Complete Deletion of <i>Slc52a2</i> Causes Embryonic Lethality in Mice-Reference-Cited by-同舟云学术

Complete Deletion of Slc52a2 Causes Embryonic Lethality in Mice

Published:2021-02-01 Issue:2 Volume:44 Page:283-286
ISSN:0918-6158
Container-title:Biological and Pharmaceutical Bulletin
language:en
Short-container-title:Biol. Pharm. Bull.

Author:

Jin Congyun¹²,Matsui Yoshihiro¹²,Yonezawa Atsushi¹²,Imai Satoshi¹,Ogihara Takashi¹²,Itohara Kotaro¹,Nakagawa Shunsaku¹,Nakagawa Takayuki¹,Matsubara Kazuo¹

Affiliation:

1. Department of Clinical Pharmacology and Therapeutics, Kyoto University Hospital

2. Graduate School of Pharmaceutical Sciences, Kyoto University

Publisher

Pharmaceutical Society of Japan

Subject

Pharmaceutical Science,Pharmacology,General Medicine

Link

https://www.jstage.jst.go.jp/article/bpb/44/2/44_b20-00751/_pdf

Reference14 articles.

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2. 2) Powers HJ, Corfe BM, Nakano E. Riboflavin in development and cell fate. Subcell. Biochem., 56, 229–245 (2012).

3. 3) Yonezawa A, Inui K. Novel riboflavin transporter family RFVT/SLC52: identification, nomenclature, functional characterization and genetic diseases of RFVT/SLC52. Mol. Aspects Med., 34, 693–701 (2013).

4. 4) Yao Y, Yonezawa A, Yoshimatsu H, Masuda S, Katsura T, Inui K. Identification and comparative functional characterization of a new human riboflavin transporter hRFT3 expressed in the brain. J. Nutr., 140, 1220–1226 (2010).

5. 5) Foraker AB, Khantwal CM, Swaan PW. Current perspectives on the cellular uptake and trafficking of riboflavin. Adv. Drug Deliv. Rev., 55, 1467–1483 (2003).

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