Ginsenoside Rh2 Inhibited Proliferation by Inducing ROS Mediated ER Stress Dependent Apoptosis in Lung Cancer Cells-Reference-Cited by-同舟云学术

Ginsenoside Rh2 Inhibited Proliferation by Inducing ROS Mediated ER Stress Dependent Apoptosis in Lung Cancer Cells

Published:2017 Issue:12 Volume:40 Page:2117-2124
ISSN:0918-6158
Container-title:Biological & Pharmaceutical Bulletin
language:en
Short-container-title:Biol. Pharm. Bull.

Author:

Ge Guanqun¹,Yan Yan²,Cai Hui³

Affiliation:

1. Department of Breast Surgery, The First Affiliated Hospital of Xi’an Jiaotong University

2. The Second Department of Thoracic Surgery, The First Affiliated Hospital of Xi’an Jiaotong University

3. Department of Vascular Surgery, The First Affiliated Hospital of Xi’an Jiaotong University

Publisher

Pharmaceutical Society of Japan

Subject

Pharmaceutical Science,Pharmacology,General Medicine

Link

https://www.jstage.jst.go.jp/article/bpb/40/12/40_b17-00463/_pdf

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3. 3) Pasini F, Verlato G, Durante E, de Manzoni G, Valduga F, Accordini S, Pedrazzani C, Terzi A, Pelosi G. Persistent excess mortality from lung cancer in patients with stage I non-small-cell lung cancer, disease-free after 5 years. Br. J. Cancer, 88, 1666–1668 (2003).

4. 4) Wang G, Yang ZQ, Zhang K. Endoplasmic reticulum stress response in cancer: molecular mechanism and therapeutic potential. Am. J. Transl. Res., 2, 65–74 (2010).

5. 5) Wang WA, Groenendyk J, Michalak M. Endoplasmic reticulum stress associated responses in cancer. Biochim. Biophys. Acta, 1843, 2143–2149 (2014).

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