TJN-331 Improves Anti-glomerular Basement Membrane Nephritis by Inhibiting the Production of Intraglomerular Transforming Growth Factor-.BETA.1
Author:
Affiliation:
1. Tsumura Research Laboratories, Tsumura & Co.
2. Department of Pathophysiology and Therapeutics, Division of Pharmasciences, Faculty of Pharmaceutical Sciences, Hokkaido University
3. Iwaki Meisei University
Publisher
Pharmaceutical Society of Japan
Subject
Pharmaceutical Science,Pharmacology,General Medicine
Link
http://www.jstage.jst.go.jp/article/bpb/33/8/33_8_1349/_pdf
Reference16 articles.
1. 1) Suzuki Y., Ogawa T., Ito M., Nagamatsu T., Pharmacometric, 19, 247—257 (1980).
2. Antibody blockade of TNF-α reduces inflammation and scarring in experimental crescentic glomerulonephritis
3. Acteoside, a Component of Stachys Sieboldii MIQ, May Be a Promising Antinephritic Agent (3): Effect of Acteoside on Expression of Intercellular Adhesion Molecule-1 in Experimental Nephritic Glomeruli in Rats and Cultured Endothelial Cells
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1. The promoter region (G-800A and C-509T) polymorphisms of transforming growth factor-β1 gene among young women with recurrent urinary tract infection;Egyptian Journal of Medical Human Genetics;2014-04
2. Transforming growth factor-beta and the glomerular filtration barrier;Kidney Research and Clinical Practice;2013-03
3. Effect of TJN-331 on Anti-Thy1 Nephritis in Rats via Inhibition of Transforming Growth Factor-.BETA.1 Production;Biological and Pharmaceutical Bulletin;2010
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