Sulphur Antioxidants Inhibit Oxidative Stress Induced Retinal Ganglion Cell Death by Scavenging Reactive Oxygen Species but Influence Nuclear Factor (Erythroid-Derived 2)-Like 2 Signalling Pathway Differently

Author:

Majid Aman Shah Abdul12,Yin Zheng Qin3,Ji Dan13

Affiliation:

1. Nuffield Laboratory of Ophthalmology, University of Oxford, John Radcliffe Hospital

2. Advanced Medical and Dental Institute, Universiti Sains Malaysia

3. Key Lab of Visual Damage and Regeneration & Restoration of Chongqing, Southwest Eye Hospital, Southwest Hospital, The Third Military Medical University

Publisher

Pharmaceutical Society of Japan

Subject

Pharmaceutical Science,Pharmacology,General Medicine

Reference63 articles.

1. 1) Quigley HA. Neuronal death in glaucoma. Prog. Retin. Eye Res., 18, 39–57 (1999).

2. 2) Sen CK. Redox signaling and the emerging therapeutic potential of thiol antioxidants. Biochem. Pharmacol., 55, 1747–1758 (1998).

3. 3) Butterfield DA, Drake J, Pocernich C, Castegna A. Evidence of oxidative damage in Alzheimer’s disease brain: central role for amyloid β-peptide. Trends Mol. Med., 7, 548–554 (2001).

4. 4) Maher P, Hanneken A. The molecular basis of oxidative stress-induced cell death in an immortalized retinal ganglion cell line. Invest. Ophthalmol. Vis. Sci., 46, 749–757 (2005).

5. 5) Brand MD, Affourtit C, Esteves TC, Green K, Lambert AJ, Miwa S, Pakay JL, Parker N. Mitochondrial superoxide: production, biological effects, and activation of uncoupling proteins. Free Radic. Biol. Med., 37, 755–767 (2004).

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