The potential of sestrins as therapeutic targets for diabetes
Author:
Publisher
Informa Healthcare
Subject
Clinical Biochemistry,Drug Discovery,Pharmacology,Molecular Medicine
Link
http://www.tandfonline.com/doi/pdf/10.1517/14728222.2015.1044976
Reference17 articles.
1. Regeneration of Peroxiredoxins by p53-Regulated Sestrins, Homologs of Bacterial AhpD
2. Sestrins Activate Nrf2 by Promoting p62-Dependent Autophagic Degradation of Keap1 and Prevent Oxidative Liver Damage
3. Sestrin 2 Is Not a Reductase for Cysteine Sulfinic Acid of Peroxiredoxins
4. p53 Target Genes Sestrin1 and Sestrin2 Connect Genotoxic Stress and mTOR Signaling
5. Sestrins Function as Guanine Nucleotide Dissociation Inhibitors for Rag GTPases to Control mTORC1 Signaling
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2. Evaluation of sestrin 2 and tribbles homolog 3 levels in obese and nonobese women with polycystic ovary syndrome;Turkish Journal of Medical Sciences;2023-12-12
3. Circular RNA SESN2 aggravates gestational trophoblast cell damage induced by high glucose by binding to IGF2BP2;Molecular Reproduction and Development;2023-01-09
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5. The functions and roles of sestrins in regulating human diseases;Cellular & Molecular Biology Letters;2022-01-03
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