Intracellular immune dysfunction in myalgic encephalomyelitis/chronic fatigue syndrome: state of the art and therapeutic implications
Author:
Publisher
Informa Healthcare
Subject
Clinical Biochemistry,Drug Discovery,Pharmacology,Molecular Medicine
Link
http://www.tandfonline.com/doi/pdf/10.1517/14728222.12.3.281
Reference67 articles.
1. Immunologic Aspects of Chronic Fatigue Syndrome
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3. CHRONIC FATIGUE SYNDROME AND FIBROMYALGIA
4. The 2–5 A system: Modulation of viral and cellular processes through acceleration of RNA degradation
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1. Naltrexone Restores Impaired Transient Receptor Potential Melastatin 3 Ion Channel Function in Natural Killer Cells From Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients;Frontiers in Immunology;2019-10-31
2. Validation of impaired Transient Receptor Potential Melastatin 3 ion channel activity in natural killer cells from Chronic Fatigue Syndrome/ Myalgic Encephalomyelitis patients;Molecular Medicine;2019-04-23
3. Chronic viral infections in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS);Journal of Translational Medicine;2018-10-01
4. Loss of Transient Receptor Potential Melastatin 3 ion channel function in natural killer cells from Chronic Fatigue Syndrome/Myalgic Encephalomyelitis patients;Molecular Medicine;2018-08-14
5. Rituximab impedes natural killer cell function in Chronic Fatigue Syndrome/Myalgic Encephalomyelitis patients: A pilot in vitro investigation;BMC Pharmacology and Toxicology;2018-03-27
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