Sp1-Induced SETDB1 Overexpression Transcriptionally Inhibits HPGD in a β-Catenin-Dependent Manner and Promotes the Proliferation and Metastasis of Gastric Cancer-Reference-Cited by-同舟云学术

Sp1-Induced SETDB1 Overexpression Transcriptionally Inhibits HPGD in a β-Catenin-Dependent Manner and Promotes the Proliferation and Metastasis of Gastric Cancer

Published:2022 Issue:4 Volume:22 Page:319
ISSN:2093-582X
Container-title:Journal of Gastric Cancer
language:en
Short-container-title:J Gastric Cancer

Author:

Fan Yaguan¹²^ORCID,Yang Libo¹²^ORCID,Ren Yi¹²^ORCID,Wu Yunhua¹²^ORCID,Li Linhai³⁴^ORCID,Li Lihua¹²^ORCID

Affiliation:

1. Department of Medical Oncology, The First People’s Hospital of Yunnan Province, Kunming, P.R. China.

2. Department of Medical Oncology, Kunming University of Science and Technology School of Medicine, Kunming, P.R. China.

3. Department of General Surgery, The First People’s Hospital of Yunnan Province, Kunming, P.R. China.

4. Department of General Surgery, Kunming University of Science and Technology School of Medicine, Kunming, P.R. China.

Funder

National Science Fund Subsidized Project

Yunnan Provincial Science and Technology Department

Publisher

The Korean Gastric Cancer Association

Subject

Cancer Research,Gastroenterology,Oncology

Link

https://jgc-online.org/pdf/10.5230/jgc.2022.22.e26

Reference40 articles.

1. TTC36 inactivation induce malignant properties via Wnt-β-catenin pathway in gastric carcinoma

2. Targeted Therapy in Advanced Gastric Carcinoma: The Future is Beginning

3. miR-21-mediated regulation of 15-hydroxyprostaglandin dehydrogenase in colon cancer

4. 15-Deoxy-Δ12,14-prostaglandin J2 Upregulates the Expression of 15-Hydroxyprostaglandin Dehydrogenase by Inducing AP-1 Activation and Heme Oxygenase-1 Expression in Human Colon Cancer Cells

5. Regulation of Transcription Factor SP1 by the β-Catenin Destruction Complex Modulates Wnt Response

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