High mobility group box 1 (HMGB1) acts as an “alarmin” to promote acute myeloid leukaemia progression-Reference-Cited by-同舟云学术

High mobility group box 1 (HMGB1) acts as an “alarmin” to promote acute myeloid leukaemia progression

Published:2018-02-27 Issue:6 Volume:7 Page:e1438109
ISSN:2162-402X
Container-title:OncoImmunology
language:en
Short-container-title:OncoImmunology

Author:

Yasinska Inna M.¹,Gonçalves Silva Isabel¹,Sakhnevych Svetlana S.¹,Ruegg Laura¹,Hussain Rohanah²,Siligardi Giuliano²,Fiedler Walter³,Wellbrock Jasmin³,Bardelli Marco⁴,Varani Luca⁴,Raap Ulrike⁵,Berger Steffen⁶,Gibbs Bernhard F.¹⁵,Fasler-Kan Elizaveta⁶⁷,Sumbayev Vadim V.¹

Affiliation:

1. Medway School of Pharmacy, Universities of Kent and Greenwich, Chatham Maritime, United Kingdom

2. Beamline 23, Diamond Light Source, Didcot, UK

3. Department of Oncology, Hematology and Bone Marrow Transplantation with Section Pneumology, Hubertus Wald University Cancer Center, University Medical Center Hamburg-Eppendorf, Germany

4. Institute for Research in Biomedicine, Universita' della Svizzera italiana (USI), Bellinzona, Switzerland

5. Department of Medicine (Dermatology and Allergology), University of Oldenburg, Germany

6. Department of Pediatric Surgery and Department of Biomedical Research, Children's Hospital, Inselspital, University of Bern, Switzerland

7. Department of Biomedicine, University Hospital Basel and University of Basel, Basel, Switzerland

Publisher

Informa UK Limited

Subject

Oncology,Immunology,Immunology and Allergy

Link

https://www.tandfonline.com/doi/pdf/10.1080/2162402X.2018.1438109

Reference31 articles.

1. The dual role and therapeutic potential of high-mobility group box 1 in cancer

2. Tumor-infiltrating DCs suppress nucleic acid–mediated innate immune responses through interactions between the receptor TIM-3 and the alarmin HMGB1

3. HMGB1 in Cancer: Good, Bad, or Both?

4. Many faces of DAMPs in cancer therapy

5. Upregulation of miR-142-3p Improves Drug Sensitivity of Acute Myelogenous Leukemia through Reducing P-Glycoprotein and Repressing Autophagy by Targeting HMGB1

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