Agraphia in patients with frontotemporal dementia and parkinsonism linked to chromosome 17 with P301LMAPTmutation: dysexecutive, aphasic, apraxic or spatial phenomenon?-Reference-Cited by-同舟云学术

Agraphia in patients with frontotemporal dementia and parkinsonism linked to chromosome 17 with P301LMAPTmutation: dysexecutive, aphasic, apraxic or spatial phenomenon?

Published:2012-11-05 Issue:1 Volume:20 Page:69-86
ISSN:1355-4794
Container-title:Neurocase
language:en
Short-container-title:Neurocase

Author:

Sitek Emilia J.,Narozanska Ewa,Barczak Anna,Jasinska-Myga Barbara,Harciarek Michał,Chodakowska-Zebrowska Małgorzata,Kubiak Małgorzata,Wieczorek Dariusz,Konieczna Seweryna,Rademakers Rosa,Baker Matt,Berdynski Mariusz,Brockhuis Bogna,Barcikowska Maria,Zekanowski Cezary,Heilman Kenneth M.,Wszolek Zbigniew K.,Slawek Jarosław

Publisher

Informa UK Limited

Subject

Clinical Neurology,Arts and Humanities (miscellaneous)

Link

http://www.tandfonline.com/doi/pdf/10.1080/13554794.2012.732087

Reference60 articles.

1. Frontotemporal dementia: impact of P301L tau mutation on a healthy carrier

2. DYSEXECUTIVE AGRAPHIA: A MAJOR EXECUTIVE DYSFUNCTION SIGN

3. Clinical-pathologic study of biomarkers in FTDP-17 (PPND family with N279K tau mutation)

4. Mutations in progranulin cause tau-negative frontotemporal dementia linked to chromosome 17

5. A clinical pathological comparison of three families with frontotemporal dementia and identical mutations in the tau gene (P301L)

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