Quantitative evaluation in vivo of the degree of differentiation of hindlimb cartilage in a rat clubfoot model
Author:
Publisher
Informa UK Limited
Subject
Health, Toxicology and Mutagenesis,Toxicology
Link
http://www.tandfonline.com/doi/pdf/10.1080/15376510802609737
Reference21 articles.
1. Retinoid Receptor Antagonists Alter the Pattern of Apoptosis in Organogenesis Stage Mouse Limbs
2. Retinoic-acid-induced limb-reduction defects: Perturbation of zones of programmed cell death as a pathogenetic mechanism
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4. Retinoic Acid Is a Potent Regulator of Growth Plate Chondrogenesis
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1. All-Trans-Retinoid Acid (ATRA) Activates Notch Signaling to Inhibit Hind Limb Chondrogenesis by Suppressing Differentiation of Chondrogenic Cells;Biology Bulletin;2023-12
2. Genotype-phenotype correlation in clubfoot (talipes equinovarus);Journal of Medical Genetics;2021-11-15
3. All-trans-retinoic acid suppresses rat embryo hindlimb bud mesenchymal chondrogenesis by modulating HoxD9 expression;Bioengineered;2021-01-01
4. All-trans-retinoic acid inhibits chondrogenesis of rat embryo hindlimb bud mesenchymal cells by downregulating p53 expression;Molecular Medicine Reports;2015-03-04
5. All-trans-retinoid acid (ATRA) suppresses chondrogenesis of rat primary hind limb bud mesenchymal cells by downregulating p63 and cartilage-specific molecules;Environmental Toxicology and Pharmacology;2014-09
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