Knockdown of KCNQ1OT1 Alleviates the Activation of NLRP3 Inflammasome Through miR-17-5p/TXNIP Axis in Retinal Müller Cells-Reference-Cited by-同舟云学术

Knockdown of KCNQ1OT1 Alleviates the Activation of NLRP3 Inflammasome Through miR-17-5p/TXNIP Axis in Retinal Müller Cells

Published:2024-08-05 Issue: Volume: Page:1-10
ISSN:0271-3683
Container-title:Current Eye Research
language:en
Short-container-title:Current Eye Research

Author:

Liu Yu¹,Zhu Manhui²,Dou Yuping¹,Xue Aihua¹,Chen Xiujuan¹,Leng Kai³,Dong Lili¹,Cao Guoping¹

Affiliation:

1. Department of Ophthalmology, The Affiliated Taizhou People’s Hospital of Nanjing Medical University, Taizhou School of Clinical Medicine, Nanjing Medical University, Taizhou, Jiangsu, China

2. Department of Ophthalmology, Lixiang Eye Hospital of Soochow University, Suzhou, Jiangsu, China

3. Department of Medical Informatics, Jiangsu Province Hospital, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China

Funder

Taizhou People’s Hospital Scientific Research Fund Project

Gusu Health Talent Program Project in Suzhou

Suzhou Science and Technology Bureau

Publisher

Informa UK Limited

Link

https://www.tandfonline.com/doi/pdf/10.1080/02713683.2024.2378037

Reference32 articles.

1. Novelties in Diabetic Retinopathy

2. A prodrug of epigallocatechin-3-gallate alleviates high glucose-induced pro-angiogenic factor production by inhibiting the ROS/TXNIP/NLRP3 inflammasome axis in retinal Müller cells

3. Thioredoxin interacting protein (TXNIP) induces inflammation through chromatin modification in retinal capillary endothelial cells under diabetic conditions

4. TXNIP Links Innate Host Defense Mechanisms to Oxidative Stress and Inflammation in Retinal Muller Glia under Chronic Hyperglycemia: Implications for Diabetic Retinopathy

5. Critical role of TXNIP in oxidative stress, DNA damage and retinal pericyte apoptosis under high glucose: Implications for diabetic retinopathy