Thalidomide and Thalidomide Transformed by pH-Dependent Hydrolysis or by Liver Enzyme Treatment Does Not Impede the Proliferation of Endothelial Cells-Reference-Cited by-同舟云学术

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Thalidomide and Thalidomide Transformed by pH-Dependent Hydrolysis or by Liver Enzyme Treatment Does Not Impede the Proliferation of Endothelial Cells

Published:2008-01 Issue:2 Volume:30 Page:307-316
ISSN:0892-3973
Container-title:Immunopharmacology and Immunotoxicology
language:en
Short-container-title:Immunopharmacology and Immunotoxicology

Author:

Shannon E.J.,Sandoval F.G.

Publisher

Informa UK Limited

Subject

Pharmacology,Toxicology,General Medicine,Immunology,Immunology and Allergy

Link

http://www.tandfonline.com/doi/pdf/10.1080/08923970801925406

Reference18 articles.

1. Thalidomide in Cancer

2. Effects of Putative Hydroxylated Thalidomide Metabolites on Blood Vessel Density in the Chorioallantoic Membrane (CAM) Assay and on Tumor and Endothelial Cell Proliferation.

3. Thalidomide is an inhibitor of angiogenesis.

4. Hydrolysis of thalidomide abrogates its ability to enhance mononuclear cell synthesis of IL-2 as well as its ability to suppress the synthesis of TNF-α

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Thalidomide delayed the ability of 4T1 cells to amass into tumors in Balb/c mice;Immunopharmacology and Immunotoxicology;2011-09-27

2. Thalidomide suppressed the growth of 4T1 cells into solid tumors in Balb/c mice in a combination therapy with the oncolytic fusogenic HSV-1 OncdSyn;Cancer Chemotherapy and Pharmacology;2009-03-24

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