Molecular Mechanism of Potentially Lethal Damage Repair. I. Enhanced Fidelity of DNA Double-strand Break Rejoining under Conditions Allowing Potentially Lethal Damage Repair
Author:
Publisher
Informa UK Limited
Subject
Radiology Nuclear Medicine and imaging,Radiological and Ultrasound Technology
Link
http://www.tandfonline.com/doi/pdf/10.1080/09553009514550331
Reference38 articles.
1. The REV2 gene of Saccharomyces cerevisiae: cloning and DNA sequence
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1. Comparison of the repair of potentially lethal damage after low- and high-LET radiation exposure, assessed from the kinetics and fidelity of chromosome rejoining in normal human fibroblasts;Journal of Radiation Research;2013-05-14
2. The effect of DN (dominant-negative) Ku70 and reoxygenation on hypoxia cell-kill: Evidence of hypoxia-induced potentially lethal damage;International Journal of Radiation Biology;2012-05-31
3. A Comparison of Chromosome Repair Kinetics in G0and G1Reveals that Enhanced Repair Fidelity under Noncycling Conditions Accounts for Increased Potentially Lethal Damage Repair;Radiation Research;2010-11
4. Repair of Potentially Lethal Damage in Normal Cells and Ataxia Telangiectasia Cells; Consideration of Non-Homologous End-Joining;Journal of Radiation Research;2007
5. Cisplatin-mediated DNA double-strand breaks in replicating but not in quiescent cells of the yeast Saccharomyces cerevisiae;Toxicology;2005-09
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